Functional Assessment Report
Summarization of Case
Luther, 80 years of age, was admitted to a nursing hospital due to Alzheimer's disease complications. As a farmer, Luther spent most of his life moving freely unlike what he was experiencing in the nursing home. However, he had to adapt to the new life. Despite his impairment memory problem, Luther was psychically fit as he managed to walk around the nursing premises. Later, he started experienced a problem in that he walked alone. For safety reasons, he was not allowed to go outside alone. Despite the warnings, he managed to walk go outside alone, even during cold periods without a coat. Each time he went out, the nursing staff had to bring him back. The architecture of the nursing home depicts one main door at the station, another one near the business office, and three fire doors at the sides and back of the building. The four wings of the building form a square, enclosing the courtyard at the center of the building.
Interview Summarization
After the interview with the staff members who work closely with Luther, I realized various that most of his actions were because of the Alzheimer's disease. Before visiting the hospital, Luther's wife ascertained that he experienced issues dealing with memory loss, an aspect that made it difficult for him to remember newly learned information. Staff members also acknowledged the condition was not because of old age, as he was not old. Luther actively conversed with staff members and managed to walk depicting his physical fitness. The recent behaviors were associated with the deepening condition of Alzheimer's disease.
Even before he was transferred to the nursing home, staff members observed signs of increased forgetfulness on the instructions given to him. One staff member acknowledged that the structure of the nursing home made it difficult for Luther to adapt to the new environment. The four hallways forming a squared structured may have occurred as confusing events regarding the aspects of time and place within this home. Under the new environment, staff members acknowledge the possibility that the structure may have deepened his confusion about the place, an aspect that explains his behavior of frequently walking out of his room. However, another interviewed staff member related Luther's behavior of frequent walking out from him room to the new environment, as he tried to learn and understand it.
ABC Data
A prominent aspect of Alzheimer's disease depicts cognitive decline, a common sign of memory impairment. In Luther's case, it is evident that AD's symptoms such as confusion, serious memory loss, disorientation, mood and behavioral changes have become common (Lu & Bludau, 2011). With the progress of time, the symptoms seem to worsen as Luther frequently leaves his room, even at the cold period, forgetting the need of a coat. The symptoms clearly depict failing brain cells. Changes in the cerebral cortex result in memory, language, visual, and learning deficits. Cognitive dysfunction explains the distribution of AD pathological changes. The changes vary based on the severity of AD. Further, the changes depend on the position of the disease regarding the spectrum of dementia.
AD related disorders such as the Parkinson's disease and Lewy body dementia (LBD) depicts similar symptoms of cognitive decline. Additionally, the disorders also share pathological features that including the degeneration of cortical cholinergic (Lu & Bludau, 2011). Unequal distribution of cognitive deficit in vascular dementia (VaD) reflects the severe impairment of some body functions. Physiological correlates of cholinergic dysfunction in the brain encompass regional glucose metabolism and cerebral blood flow. Vital efficacy parameters in AD include direct assessment of cognitive improvement and stabilization. Efficacy in the treatment of AD has encompassed studies in ChE inhibitors that carry along benefits in long-term treatment on the improvement of cognitive failure of AD (Lu & Bludau, 2011).
Section 2
Hypothesis
Amyloid precursor protein (APP) is a common protein found within the bodies of humans. The amyloid hypothesis depicts that a fault in processing the amyloid precursor protein (APP) within the human brain propels the production of the beta-amyloid, a short fragment of APP (Lu & Bludau, 2011). Accumulation of the beta amyloid in the brain triggers disruption and destruction of nerve cells. As a result, Alzheimer's diseases occur. Amyloid plaques refer to the accumulated clumps of beta amyloid. Fault regarding the overproduction of beta-amyloid results in the occurrence of AD. Additionally, the fault in the mechanism that clears beta amyloid from the brain also explains AD.
Functional Analysis Conditions
In this analysis, we utilize an "alternative design," also referred to as a "multi-element design" (Miltenberger, 2015). The design depicts the utilization of one particular type of manipulation for a given day or session. Consequent...
Our semester plans gives you unlimited, unrestricted access to our entire library of resources —writing tools, guides, example essays, tutorials, class notes, and more.
Get Started Now